Congratulations to Scott Williams’s fourth-year graduate student Kendall Lough, who was recently awarded a “Ruth L. Kirschstein National Research Service Award (NRSA) Individual Predoctoral Fellowship (F31)” from the NIH/NIDCR (National Institute of Dental and Craniofacial Research). Kendall is researching the role of nectin adhesion proteins and their downstream effector afadin in palate closure.
Another outstanding undergraduate in the Scott Williams lab, Jeet Patel, who is completing his Senior Honors Thesis this spring, had his research supported by a “Tom and Elizabeth Long Research Award”. Jeet also participated in a competitive research internship at the Harvard Center for Systems Biology last summer. He will be attending graduate school at the University of Washington where he will be hopes to use computational and systems approaches to study stem cells and development. Jeet will be getting a head start on his studies by doing his first rotation in Cecilia Moens lab working on neuronal migration in zebrafish.
Jeff Dangl’s lab members Gabriel Castrillo, Paulo Teixeira, and Sur Herrera Paredes led a study with current Dangl lab members Terry Law and Omri Finkel and other collaborators which was published in Nature online on March 15. The article “Root microbiota drive direct integration of phosphate stress and immunity” shows that microbes growing with plants affect both immune responses and nutrient starvation responses by activating a common transcription factor that upregulates starvation response genes and represses immune signaling genes. READ THE PAPER>> READ MORE >>
Postdocs Li Yang and Paulo Teixeira (Jeff Dangl’s lab) are co-first authors along with current Dangl lab members Omri Finkel and Isai Salas-Gonzalez, former Biology undergraduate student Marie English and other collaborators on the paper titled “Pseudomonas syringae Type III effector HopBB1 promotes host transcriptional repressor degradation to regulate phytohormone responses and virulence” published in Cell Host and Microbe Feb. 8, 2017. The researchers show that a bacterial virulence protein suppresses plant immune responses by disrupting the interaction of transcription factors associated with plant hormones that affect growth and defense. READ MORE >>